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LA

lansoprazole + amoxicillin + clarithromycin (Lansap)

✓ Approved

Takeda · ATP4A · Small Molecule

What is lansoprazole + amoxicillin + clarithromycin?

lansoprazole + amoxicillin + clarithromycin is a small molecule developed by Takeda. It is approved for therapeutic indications via oral (po).

Drug Profile

Brand NamesLansap
CompanyTakeda
Drug ClassSmall Molecule
Molecular TargetATP4A
RouteOral (PO)
StatusApproved

Mechanism of Action

Molecular Targets

lansoprazole + amoxicillin + clarithromycin acts on 1 molecular target:

ATP4AATPase H+/K+ transporting subunit alpha (ATP6A)
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Therapeutic Indications

lansoprazole + amoxicillin + clarithromycin is developed for 2 unique indications across 2 therapeutic areas.

Therapeutic AreaConditionPhase
Gastrointestinal disordersGastric ulcer✓ Approved
Infections and infestationsHelicobacter infection✓ Approved

Related Research Articles

PubMedMedicine2026-07-25

Helicobacter pylori antibiotic resistance genes and virulence based on fecal samples in Qinghai, China: A cross-sectional study.

Wei Shenghui S, Wang Xuehong X, Xu Ling L

Antibiotic resistance in Helicobacter pylori (H pylori) is influenced by multiple factors and shows significant geographical variation. Qinghai has a high incidence of gastric cancer and a high prevalence of H pylori infection, which may be exacerbated by factors such as limited healthcare resources, an underdeveloped surveillance system, a challenging geographical environment and climate, and distinct local lifestyle habits. This study aimed to investigate the distribution of resistance genes and virulence factors of H pylori strains isolated from patients in the Qinghai region to levofloxacin, clarithromycin, amoxicillin, tetracycline, and furazolidone, in order to provide a scientific basis for the precise treatment of H pylori in Qinghai. Between October 2024 and August 2025, 151 patients in Qinghai province who underwent 13C-urea breath testing for H pylori infection were enrolled. The overall antibiotic resistance rate of H pylori in Qinghai province was 80.60%, with the highest rate observed for clarithromycin (58.21%) and the lowest for tetracycline (14.18%). Resistance rates for levofloxacin, amoxicillin, and furazolidone were 41.04%, 17.91%, and 17.91%, respectively. The rates of dual and multidrug resistance were 31.34% and 17.91%, respectively. Antibiotic resistance in H pylori from Qinghai poses a significant challenge, with a concomitantly high detection rate of virulence factors. Therefore, it is imperative to proactively implement antibiotic susceptibility testing and assess relevant influence factors for H pylori-infected patients in Qinghai prior to the 1st eradication therapy to reduce antibiotic resistance.

PubMedData in brief2026-07-25

Dataset of genome sequencing and amoxicillin degradation data of Bacillus cereus BT-C2.4 isolated from aquaculture water.

Luu Hong Lat HL, Phan Nhu Nguyet NN, Lam Thanh Nhan TN, Phan Trang Thi Phuong TTP

This dataset describes the draft genome sequence and experimental amoxicillin degradation data of B. cereus BT-C2.4, isolated from aquaculture water in Ben Tre province, Vietnam. The genome was sequenced using Illumina technology and assembled into 61 contigs with a total length of approximately 5.44 Mb and GC content of 35%. Genome annotation was performed using the NCBI Prokaryotic Genome Annotation Pipeline (PGAP). The dataset also includes high performance liquid chromatography (HPLC) data measuring residual amoxicillin concentrations over time, showing that amoxicillin was fully degraded within 24 h under the tested conditions. These data may be useful for studies on antimicrobial resistance, antibiotic biodegradation, and environmental microbiology in aquaculture systems.

PubMedMedicine2026-07-25

Proton pump inhibitors use and the risk of osteoporosis and fractures: A two-sample Mendelian randomization study.

Li Ping P, Wu Ruiji R, Shi Hangchu H

The causal relationship between proton pump inhibitor (PPI) use and bone health outcomes remains uncertain. This study employs a Mendelian randomization (MR) approach to investigate the potential causal association between PPI use and the risk of osteoporosis and fractures. We selected 4 representative PPIs, including omeprazole, esomeprazole, lansoprazole, and rabeprazole, for our study. Bone health outcomes were evaluated through femoral neck bone mineral density (BMD), lumbar spine BMD, and the prevalence of osteoporosis and fracture across various anatomical sites, including the upper arm and shoulder, wrist and hand, lumbar spine and pelvis, femur, and lower leg and ankle. To evaluate PPI exposure and bone health outcomes, we utilized summary statistics derived from genome-wide association studies conducted in European ancestry populations. Primary causal estimates were derived using the inverse-variance weighting (IVW) approach, supplemented by MR-Egger, weighted median, and Mendelian Randomization Pleiotropy Residual Sum and Outlier methods. To strengthen the robustness of our findings, we conducted sensitivity analyses encompassing assessments of heterogeneity, horizontal pleiotropy, and leave-one-out analyses. Lansoprazole demonstrated a significant positive causal effect on femoral neck BMD (IVW: β = 0.137, 95% confidence interval: 0.063-0.210, P = 2.73E-04), whereas no statistically significant effects were observed for lansoprazole on lumbar spine BMD, osteoporosis, or fracture risk. Esomeprazole showed a marginal causal association with an elevated risk of femur fracture (IVW: odds ratio = 1.049, 95% confidence interval: 1.004-1.096, P = .031); however, this association lost statistical significance following Bonferroni correction. Its effects on BMD, osteoporosis, and fractures at other anatomical sites remained nonsignificant. No causal associations with BMD, osteoporosis, or fracture risk were identified for either omeprazole or rabeprazole. Sensitivity analyses further reinforced the robustness and reliability of these findings. This MR analysis found no compelling evidence to support a causal association between PPI use and the risk of osteoporosis or fractures.

PubMedThe Journal of dermatology2026-07-24

Pregnancy-Associated Pityriasis Lichenoides et Varioliformis Acuta Responsive to Clarithromycin.

Hosoi Misato M, Takahashi Miho M, Maru Reina R, Tsuchihashi Hitoshi H et al.

PubMedLancet (London, England)2026-07-24

Oral step-down, optimal drug, and total duration of antibiotic treatment in African children hospitalised with severe community-acquired pneumonia (PediCAP): a factorial randomised controlled trial.

Bielicki Julia A JA, Clements Michelle M, Musiime Victor V, Moore David P DP et al.

WHO recommends 5 days of intravenous antibiotics for children hospitalised with severe community-acquired pneumonia (CAP). We aimed to investigate the safety of a step-down to different oral antibiotics and the shortest effective duration. PediCAP was an open-label, parallel group, 2 × 5 factorial randomised controlled trial of children aged 2 months to 6 years hospitalised with severe CAP without complicating factors in 13 hospitals across five sub-Saharan African countries. Children weighing 3-30 kg and with point-of-care C-reactive protein of more than 10 mg/L were randomly assigned (5:5:1) to either a step-down from intravenous antibiotics to oral amoxicillin (250 mg) or amoxicillin-clavulanate (co-amoxiclav; 200 mg amoxicillin to 28·5 mg clavulanate) via dispersible tablets twice daily (superiority comparison) for five total (intravenous plus oral) durations (4-8 days; non-inferiority comparison), or a 5-day, fixed-duration, intravenous-only treatment (non-inferiority comparison). Children were stepped down when clinically improved and able to take oral antibiotics. Clinicians could change antibiotics if clinically indicated. The primary outcome was the proportion of readmission or death at day 28 (non-inferiority margin vs intravenous only: +10%). The study was registered with the ISRCTN registry (ISRCTN63115131) and is complete. Between Dec 7, 2020, and Aug 14, 2023, 2248 children were screened for eligibility and 1101 were randomly allocated (480 [43·6%] girls and 621 [56·4%] boys). The primary outcome was available in 1055 (95·8%) children. For children in the step-down groups, the mean total antibiotic exposure was 6·0 days (SD 3·5) in the 4-day group, 6·8 days (4·2) in the 5-day group, 7·4 (3·6) in the 6-day group, 8·3 days (3·4) in the 7-day group, and 9·2 days (2·9) in the 8-day group (p<0·0001), with 140 (68·6%) of 204, 151 (76·3%) of 198, 167 (85·2%) of 196, 179 (89·5%) of 200, and 186 (91·6%) of 203, respectively, stepping down within their randomised duration (p<0·0001). Primary outcomes occurred in 33 (6·9%) of 475 in the co-amoxiclav group, 27 (5·6%) of 484 in the amoxicillin group, and six (6·3%) of 96 in the intravenous-only group, with both oral step-down strategies non-inferior to the intravenous-only strategy (upper 95% CIs of 6·0 for co-amoxiclav and 5·7 for amoxicillin) and no evidence of superiority for co-amoxiclav over amoxicillin (adjusted risk difference 1·3% [95% CI -1·8 to 4·4]; p=0·40). Primary outcomes occurred in eight (4·1%) of 194 in the 4-day group, ten (5·3%) of 190 in the 5-day group, 16 (8·5%) of 188 in the 6-day group, 16 (8·3%) of 193 in the 7-day group, and ten (5·2%) of 194 in the 8-day group; all durations were non-inferior to the 8-day group (all upper 95% CIs ≤6·0%). There was no evidence of consistent differences in adverse events for oral antibiotic or duration comparisons. For antibiotic-related or serious adverse events, there was one (1·0%) in the intravenous-only group and 12 (2·5%) in the amoxicillin group (adjusted risk difference -2·3% [95% CI -4·2 to -0·3]; p=0·025), and 16 (3·4%) in the co-amoxiclav group (+0·8% [-1·2 to 2·8]; p=0·42); rates increased with randomised duration (slope estimate +0·9% [0·1 to 1·7]; p=0·032). For sub-Saharan African children hospitalised with severe CAP without complicating factors, a strategy of stepping down upon clinical improvement to oral amoxicillin after initial intravenous antibiotics, with a total treatment duration of 4-5 days, is non-inferior to WHO-recommended 5-day intravenous treatment. The Second European and Developing Countries Clinical Trials Partnership (EDCTP2).

PubMedThe Korean journal of gastroenterology = Taehan Sohwagi Hakhoe chi2026-07-24

[Evidence-Based Guidelines for the Diagnosis and Treatment of Helicobacter pylori Infection in Korea: 2025 Revised Edition].

Bang Chang Seok CS, Kang Seung Joo SJ, Lim Hyunchul H, Kim Seung Han SH et al.

Since the 2020 Korean guidelines for Helicobacter pylori treatment, clarithromycin resistance rates have risen from 17.8% to 33.3%, dual-priming oligonucleotide-based polymerase chain reaction-guided tailored therapy has been adopted, and potassium-competitive acid blockers (P-CABs) have become available. This fourth revision addressed these changes. Nine key questions were addressed through systematic review and meta-analysis. Thirteen recommendations were evaluated using a modified Delphi process involving 64 experts. Twelve recommendations achieved a first-round consensus; one required revision and achieved 73.9% agreement. Key changes included: 1) a dual-pillar strategy of tailored therapy and empirical quadruple therapy; 2) restricted use of empirical clarithromycin- based triple therapy under specific conditions; 3) removal of sequential therapy; 4) use of P-CABs as alternatives to proton pump inhibitors; 5) expansion of eradication indications to include gastric cancer prevention in H. pylori gastritis and regression of hyperplastic polyps ≤10 mm; and 6) positioning of bismuth quadruple therapy as a conditionally recommended first-line empirical option, with preference for reservation as salvage therapy, and introduction of modified bismuth quadruple therapy (addition of bismuth to conventional regimens) as an additional first-line empirical option. The revised guidelines provide updated evidence-based recommendations for the diagnosis and treatment of H. pylori infection, reflecting the rapidly changing antibiotic resistance landscape and the introduction of new diagnostic and therapeutic tools in Korea. These guidelines aim to assist clinicians, patients, policymakers, and medical educators in optimizing H. pylori management. They may differ from the current medical insurance standards and will be further revised based on emerging evidence.

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