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didanosine (didanosine EC / Videx EC)

✓ Approved

Bristol-Myers Squibb · · Small Molecule

What is didanosine?

didanosine is a small molecule developed by Bristol-Myers Squibb. It is approved for therapeutic indications via oral (po).

Drug Profile

Brand Namesdidanosine EC, Videx EC
CompanyBristol-Myers Squibb
Drug ClassSmall Molecule
RouteOral (PO)
StatusApproved

Mechanism of Action

Molecular Targets

didanosine acts on 1 molecular target:

gag-pol, HIV-1 (gag-pol)
Want deeper analysis?Noah AI can explain complex mechanisms and compare to similar drugs.

Therapeutic Indications

didanosine is developed for 1 unique indication across 1 therapeutic area.

Therapeutic AreaConditionPhase
Infections and infestationsAcquired immunodeficiency syndrome✓ Approved

Related Research Articles

PubMedBiopharmaceutics & drug disposition2026-05-30

Pharmacokinetic Assessment of Atazanavir and Favipiravir Following Echinacea Supplementation: A Controlled Herb-Drug Interaction Investigation.

Gajula Siva Nageswara Rao SNR, Kallam Jeevan Sai JS, Kanukolanu Aarika A, Korada Ganesh G et al.

Echinacea is a widely used herbal supplement and is frequently co-administered with antiviral drugs, raising concerns regarding potential herb-drug interactions. This study evaluated the effect of repeated Echinacea administration on the pharmacokinetics of favipiravir and atazanavir in rats. Male Sprague Dawley rats were administered Echinacea (250 mg/kg) or vehicle once daily for 10 days, followed by a single oral dose of favipiravir (70 mg/kg) or atazanavir (35 mg/kg). Plasma drug concentrations were quantified using validated LC-MS/MS methods, and pharmacokinetic parameters were determined by noncompartmental analysis. Echinacea co-administration did not significantly alter favipiravir exposure (Cmax: 39,677 ± 5385 vs. 36,364 ± 5379 ng/mL; AUC0-t: 144,457 ± 17,267 vs. 104,840 ± 27,001 ng h/mL) or atazanavir exposure (Cmax: 3274 ± 256 vs. 4004 ± 629 ng/mL; AUC0-t: 8962 ± 1667 vs. 10,645 ± 2949 ng h/mL). No relevant changes were observed in clearance, half-life, or mean residence time for either drug. These findings indicate that repeated Echinacea administration does not result in a pharmacokinetically relevant interaction with favipiravir or atazanavir under the investigated conditions.

PubMedJournal of autoimmunity2026-05-30

Orelabrutinib for systemic lupus erythematosus: A randomised, double-blind, placebo-controlled study.

Li Ru R, Li Xue X, Zhu Xiaoxia X, Liu Shengyun S et al.

Orelabrutinib is an oral, highly selective, irreversible inhibitor of Bruton's tyrosine kinase (BTK). Preclinical mechanistic studies have demonstrated its therapeutic potential in systemic lupus erythematosus (SLE). A multicentre, double-blind, randomised, placebo-controlled, parallel-group, phase Ib/IIa study was conducted in 11 centres in China. Patients diagnosed with SLE were randomised 1:1:1:1 to receive oral orelabrutinib at 50 mg, 80 mg, and 100 mg or placebo once daily for 12 weeks, respectively. This trial is registered with ClinicalTrials.gov, NCT04305197. Between July 9, 2020 and September 29, 2021, 60 patients were randomised, with 55 patients who completed 12 weeks of treatment. Adverse events (AEs) were mostly mild or moderate. In all evaluable patients, the SLE Response Index (SRI)-4 rates at week 12 were 50%, 62%, and 64% for orelabrutinib at 50 mg, 80 mg, and 100 mg, respectively, compared with 36% for placebo, indicating dose-dependent improvement. Among patients with baseline SLEDAI-2K > 8, significantly higher SRI-4 responses were noted with orelabrutinib at 50 mg (80%, p = 0·048), 80 mg (83%, p = 0·048), and 100 mg (100%, p = 0·029) compared to placebo (0%). SRI-6 responses at week 12 were 36%, 39%, and 21% for orelabrutinib at 50 mg, 80 mg, and 100 mg, respectively, compared with 7% for placebo. Reduced proteinuria, anti-dsDNA, IgG, and IgM and increased C4 were observed with orelabrutinib treatment. Orelabrutinib was well tolerated and potentially efficacious in patients with SLE.

PubMedJournal of the Formosan Medical Association = Taiwan yi zhi2026-05-30

Concurrent evolution of infant oral and gut microbiota implies oral-to-gut transmission.

Lai Chi-Ting CT, Lee Wei-Hsiang WH, Chang Yu-Yuan YY, Chiu Han-Mo HM et al.

Infant oral microbiome theoretically could serve as the seeding microorganisms in its own gut microbial construction from anatomical perspective. The purpose of this study was to investigate the relationship between oral and gut microbiota in early infantile life. This prospective longitudinal study included 15 healthy babies delivered in a single medical center during 2019∼2023. Stool samples were collected daily in the first 5 days after birth, while salivary and buccal mucosal samples were obtained once. All three samples continued acquisition at the end of 1st, 2nd, 4th and 6th month of age. The entire samples were 16S-rRNA sequenced. Investigation of the shared bacteria between oral and gut ecosystems was carried out. Niche differentiation started very early in the infants' gut ecosystem since the 2nd day of life. Bifidobacterium appeared scarcely in the first 5 days after birth but gradually rose in abundance since the 1st month. The compositions of the infant salivary and buccal mucosal microbiome were similar. Most of the infant gut and oral microbiota evolved independently, whereas the oral Rothia, Prevotella and Veillonella correlated with the development of their gut counterparts. Coprococcus, Prevotella, Ruminococcaceae, Faecalibacterium and Megasphaera also showed associations between saliva and gut. The optimal timing for examining and treating infant gut dysbiosis could be very early after birth. Together with the results from previous research, Ruminococcus and Veillonella deserve more investigation in the future to confirm their oral-to-gut transmission and potential effect in treating early-life intestinal dysbiosis.

PubMedVeterinary dermatology2026-05-30

Efficacy of 0.5% Tretinoin in the Treatment of Equine Aural Plaques.

de Mattos Felipe Sperandio FS, Conceição Lissandro Gonçalves LG, Schuartz Ytalo Galinari Henrique YGH, Silva Vitória Régia Melo VRM et al.

Equine aural plaques are a benign form of auricular papillomatosis caused by equine papillomavirus. Despite the efficacy of 5% imiquimod cream, a marked local reaction is frequently observed during therapy, often requiring sedation before application. The aim of this study was to evaluate the efficacy and potential adverse effects of 0.5% topical tretinoin in the treatment of equine aural plaques. We hypothesised that tretinoin would be effective, with fewer adverse effects compared to imiquimod. The study included 10 horses of both sexes, aged between 7 months and 15 years, diagnosed with unilateral or bilateral aural plaques (totalling 17 ears). This was a controlled clinical trial. Three horses underwent a pilot study, in which only one ear per animal was treated to allow for intra-animal comparison, while the remaining seven horses received treatment in both ears. Lesions were classified into three groups based on the percentage of the inner auricular surface affected. Treatment was performed with 0.5% topical tretinoin once daily until lesion stabilisation or resolution. Lesion characteristics, treatment response, adverse effects and recurrence were evaluated. Sixty-five percent of the ears exhibited an improvement of > 50% following treatment. On average, maximal lesion resolution occurred within 52.8 days (range 6-74 days). None of the horses exhibited severe sensitivity during the treatment period, allowing for ear manipulation throughout the study. The topical application of 0.5% tretinoin cream proved to be an effective and safe treatment for equine aural plaques. Further studies with a larger sample size and long-term follow-up are recommended to optimise treatment protocols.

PubMedInternational immunopharmacology2026-05-30

Andrographolide attenuates neuroinflammation and apoptosis after spinal cord injury via PI3K-Akt pathway activation.

Li Fei F, Zhao Qiannan Q, Zhang Jiaqi J, Shen Zhaoliang Z et al.

Spinal cord injury (SCI) triggers secondary injury cascades dominated by microglia-driven neuroinflammation, mitochondrial dysfunction, and neuronal apoptosis, leading to progressive tissue loss and persistent neurological deficits. Andrographolide, a diterpenoid lactone from Andrographis paniculata, has broad anti-inflammatory activity, but its therapeutic potential and mechanism in SCI remain unclear. Here, we combined network pharmacology with experimental validation to evaluate andrographolide after SCI. Candidate targets and pathways were screened in silico, prioritizing PI3K-Akt signaling. In a mouse contusion SCI model, andrographolide was administered intraperitoneally at 30 or 60 mg/kg starting 2 h after injury and then once daily for 14 consecutive days. PI3K-Akt pathway involvement was examined using intrathecal LY294002 under the 60 mg/kg andrographolide regimen. Behavioral assessments were conducted from 0 to 28 days post-injury to evaluate the sustained effects of early andrographolide treatment. In vitro, LPS-stimulated BV2 microglia were treated with andrographolide with or without LY294002, followed by cytokine assays, JC-1 analysis of mitochondrial membrane potential, and p-PI3K immunofluorescence. Network pharmacology identified overlapping genes between andrographolide-related targets and SCI-associated genes, and highlighted PI3K-Akt as a key enriched pathway. Consistent with this prediction, andrographolide dose-dependently restored PI3K/Akt/GSK3β/CREB phosphorylation and shifted apoptosis markers toward survival in vivo, with 60 mg/kg producing stronger effects, while LY294002 attenuated these molecular benefits. Using the selected regimen, andrographolide improved locomotor recovery and gait performance, reduced lesion cavitation, and preserved neuronal survival after SCI. In BV2 microglia, andrographolide increased p-PI3K, suppressed LPS-induced TNF-α/IL-1β/IL-6 release, and restored mitochondrial membrane potential, whereas LY294002 attenuated these effects. Collectively, andrographolide confers neuroprotection after SCI in association with activation of PI3K-Akt signaling, attenuation of inflammatory responses, preservation of mitochondrial function, and inhibition of apoptosis. Complementary BV2 cell experiments suggest that microglia-related inflammatory modulation may contribute to these protective effects.

PubMedThe Lancet. Infectious diseases2026-05-30

Efficacy and safety of a 4-month quabodepistat, delamanid, and bedaquiline regimen for drug-susceptible pulmonary tuberculosis: a multicentre, open-label, randomised, proof-of-concept, non-inferiority, phase 2b/c trial.

Dawson Rodney R, Diacon Andreas H AH, Variava Ebrahim E, Moloantoa Tumelo T et al.

Combined therapy with delamanid, bedaquiline, and quabodepistat (DBQ) showed potent early bactericidal activity and was well tolerated in a phase 2a, 14-day early bactericidal activity trial in participants with drug-susceptible pulmonary tuberculosis. This subsequent proof-of-concept trial evaluated the efficacy and safety of a 4-month, three-dose level regimen of DBQ in participants with drug-susceptible pulmonary tuberculosis compared with 6 months of the standard of care. This open-label, randomised, proof-of-concept, non-inferiority, phase 2b/c trial was conducted at six clinical research sites in South Africa. Adults aged 18-65 years with newly diagnosed, drug-susceptible pulmonary tuberculosis were recruited by research sites from community tuberculosis clinics. Participants were randomly assigned (1:2:2:1) by balanced block randomisation (block size six) to receive oral delamanid (300 mg once a day) and bedaquiline (400 mg once a day for 2 weeks then 200 mg three times a week) plus quabodepistat at once-daily doses of 10 mg (DBQ10 group), 30 mg (DBQ30 group), or 90 mg (DBQ90 group) for 4 months; or 6 months of RHEZ (rifampicin, isoniazid, ethambutol, and pyrazinamide for 8 weeks followed by 18 weeks of rifampin and isoniazid). All drugs were administered orally. Masking procedures were not used, although microbiology laboratory staff were masked to treatment assignment. The primary endpoints were the proportion of participants reaching sputum culture conversion by the end of the treatment period in the modified intention-to-treat (mITT) analysis set and safety in the safety analysis set. Non-inferiority, with a margin of 12%, was assessed for the primary endpoint in the mITT analysis set, comparing the pooled DBQ group and the RHEZ group using a two-sided 80% CI. This study was registered at ClinicalTrials.gov, NCT05221502, and is complete. Between April 12, 2022, and May 19, 2024, 306 individuals were screened, of whom 122 were enrolled and randomly assigned: 20 to the DBQ10 group, 42 to the DBQ30 group, 39 to the DBQ90 group, and 21 to the RHEZ group. 121 participants received at least one dose of study drug and comprised the mITT analysis set. At the end of the treatment period, the proportion of participants with sputum culture conversion was 100·0% (95% CI 83·2-100·0, all 20 participants) in the DBQ10 group, 92·9% (80·5-98·5, 39 of 42 participants) in the DBQ30 group, 97·4% (86·2-99·9, 37 of 38 participants) in the DBQ90 group, 96·0% (90·1-98·9, 96 of 100 participants) in the pooled DBQ group, and 100·0% (83·9-100·0, all 21 participants) in the RHEZ group. The comparison between the pooled DBQ group and the RHEZ group met non-inferiority for the primary endpoint (difference -4·0% [80% CI -7·4 to 3·4]). Adverse events were mostly mild to moderate, with no serious adverse events or discontinuations attributed to trial medications. Rates of adverse events of grade 3 or higher were 20% (four of 20 participants) in the DBQ10 group, 14% (six of 42 participants) in the DBQ30 group, 11% (four of 38 participants) in the DBQ90 group, and 5% (one of 21 participants) in the RHEZ group. One participant (in the DBQ90 group) died after worsening pulmonary tuberculosis with pneumonia, which was assessed as not related to study treatment. 105 (87%) of 121 participants had at least one treatment-emergent adverse event: 17 (85%) of 20 in the DBQ10 group, 37 (88%) of 42 in the DBQ30 group, 30 (79%) of 38 in the DBQ90 group, and all 21 (100%) participants in the RHEZ group. The most common treatment-emergent adverse events in the total population of 121 participants were upper respiratory tract infection (n=36, 30%), headache (n=24, 20%), and diarrhoea (n=12, 10%). In this proof-of-concept study, 4 months of DBQ showed a promising end-of-treatment non-inferiority signal versus 6 months of RHEZ and was generally well tolerated. Our results support further studies of quabodepistat as a potential component of new treatment-shortening regimens for tuberculosis. Otsuka Pharmaceutical Development & Commercialization, with partial funding from the Gates Foundation.

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