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miconazole + benzoyl peroxide (Acnidazil / Acne Creme Plus)

✓ Approved

Teva Pharmaceutical Industries Ltd. · Small Molecule · Small Molecule

What is miconazole + benzoyl peroxide?

miconazole + benzoyl peroxide is a small molecule developed by Teva Pharmaceutical Industries Ltd.. It is approved for therapeutic indications.

Drug Profile

Brand NamesAcnidazil, Acne Creme Plus
CompanyTeva Pharmaceutical Industries Ltd.
Drug ClassSmall Molecule
StatusApproved

Therapeutic Indications

miconazole + benzoyl peroxide is developed for 1 unique indication across 1 therapeutic area.

Therapeutic AreaConditionPhase
Skin and subcutaneous tissue disordersAcne✓ Approved

Related Research Articles

PubMedChemphyschem : a European journal of chemical physics and physical chemistry2026-07-25

Predictive Modeling of Bacterial Inactivation With Hydrogen Peroxide Over a Cobalt Ferrite Catalyst.

Husak Viktor V, Danyliuk Nazarii N, Roubik Hynek H, Bobrova Olena O et al.

Hydrogen peroxide activated by a cobalt ferrite catalyst generates hydroxyl radicals that effectively inactivate bacteria. A continuous-flow packed-bed reactor containing cobalt ferrite was developed and tested for bacterial disinfection. The inactivation kinetics were highly nonlinear. The classical Weibull model accurately described the initial decline in bacterial counts at low H2O2 concentration (3 mM) but lost accuracy at longer contact times and higher concentrations (6-10 mM). To improve prediction, 10 machine learning algorithms were evaluated. Gradient boosting (GB) and random forest (RF) showed the best performance and were assessed using cross-validation, error metrics, and independent validation with previously unseen data at 4 mM H2O2. GB demonstrated superior generalization, whereas RF was more accurate under moderate oxidative stress. A combined GB + RF model, based on averaging predictions from both algorithms, provided the most balanced results, with average errors below ±10% at low-to-moderate peroxide concentrations. The findings highlight the limitations of conventional kinetic models for cobalt ferrite-catalyzed peroxide disinfection and provide a predictive framework for selecting peroxide dose and contact time in continuous-flow treatment. Independent validation confirmed high predictive accuracy, with larger relative errors observed only at very low residual bacterial counts.

PubMedFrontiers in physiology2026-07-25

Sustained low micromolar hydrogen peroxide exposure induces sequential red blood cell dysfunction.

Baek Jin Hyen JH, Williams Matthew C MC, Jana Sirsendu S, Zhang Xiaoyuan X et al.

Red blood cells (RBCs) are continuously exposed to oxidative stress throughout their lifespan and during ex vivo storage. Most experimental oxidative stress models rely on supraphysiological bolus oxidant addition, yet this approach does not replicate the sustained, low-level oxidant exposure encountered physiologically. Understanding the temporal sequence of RBC oxidative injury under physiologically relevant conditions is essential for identifying early biomarkers of dysfunction and developing targeted interventions. We employed a glucose oxidase (GX)-based system to generate sustained hydrogen peroxide (H2O2) at low micromolar concentrations (0.8-8 µM) and examined the temporal progression of oxidative damage in human RBCs over 24 hours. Healthy donor RBCs exposed to GX (1-10 mU/mL) were evaluated for oxidative burden, antioxidant status, hemoglobin oxidation, relative hemoglobin release, membrane integrity/viability by calcein fluorescence, deformability by ektacytometry, phosphatidylserine (PS) externalization by Annexin V binding, and vesiculation by microscopy. Sustained H2O2 exposure induced rapid glutathione depletion (within 6 hours) followed by progressive methemoglobin formation. Single-cell analyses demonstrated a strong inverse relationship between intracellular oxidative autofluorescence and cellular dysfunction. GX induced concentration-dependent impairment of RBC deformability, with 10 mU/mL causing significant membrane rigidity and hemoglobin release indicative of membrane lysis by 24 hours. Time-dependent vesiculation and release of CD235a- and Band 3-labeled microvesicles occurred with vesiculating RBCs exhibiting higher oxidative burden than non-vesiculating cells. Notably, PS externalization was absent on both vesiculating RBCs and their shed microvesicles. These findings define a temporal hierarchy of oxidative injury under physiologically relevant conditions and demonstrate that vesiculation and PS externalization are mechanistically uncoupled under sustained oxidative stress. This model provides a framework for identifying early biomarkers of RBC dysfunction that may guide the development of targeted interventions to optimize blood storage and mitigate oxidative injury.

PubMedMicrobial pathogenesis2026-07-25

Competitive interactions between canine-derived lactic acid bacteria and Klebsiella michiganensis: Implications for probiotic potential in canine cystitis.

de Moraes Reiner Silveira RS, Teixiera Maria Eloisa ME, Núñez Teresa Alessandra de Lima TAL, Paschoal Natália Rodrigues NR et al.

The Klebsiella oxytoca complex comprises at least six bacterial species, including Klebsiella michiganensis, for which reports in veterinary medicine remain scarce, particularly in the context of urinary tract infections. This prospective in vitro study aimed to (i) molecularly characterize, using next-generation sequencing (NGS), a K. michiganensis strain isolated from canine cystitis, including its plasmid content and antimicrobial resistance gene repertoire; and (ii) identify, among six species-specific lactic acid bacteria (LAB) recovered from the urogenital tract of healthy dogs, candidate strains exhibiting in vitro antagonism against this pathogen and a probiotic phenotype compatible with future ex vivo and in vivo validation. The in vitro antagonism was performed through five bacterial competition assays: spot-on-the-lawn (SOTL), radial streak (RS), modified cross-streak (MCS), agar well diffusion (AWD), and liquid coculture (LCA) assays. The probiotic properties of LAB strains were assessed through resistance to artificial gastric juice, bile salt tolerance, hydrophobicity, growth capacity, hydrogen peroxide production, antimicrobial susceptibility, complemented by class 1 integron genes screening and KEGG functional profile characterization. NGS analysis of K. michiganensis revealed the presence of IncF and IncR plasmids and 27 antimicrobial resistance genes. KEGG orthology analysis of the LAB strains revealed strain-specific metabolic KOs in four out of the six isolates. Among the tested strains, Enterococcus faecalis showed greater inhibition in the SOTL assays, whereas Lacticaseibacillus casei exhibited significantly greater inhibitory activity in the RS (p < 0.0001). Although some E. faecalis strains showed limited inhibition in the AWD and MCS assays, both species effectively inhibited the pathogen in the LCA. All LAB strains demonstrated tolerance to gastrointestinal conditions, moderate to high hydrophobicity, and hydrogen peroxide production, with no class 1 integron genes detected. Overall, K. michiganensis was identified as a novel causative agent of canine cystitis, and LAB strains, particularly L. casei, displayed key probiotic traits, supporting its potential for therapeutic application.

PubMedFood chemistry2026-07-25

Emulsion-templated oleogel based on an OSA starch-gelatin-tannic acid ternary complex for algal oil delivery.

Miao Wenbo W, Han Junqing J, Quek Siew Young SY, McClements David Julian DJ et al.

In this study, ternary complexes composed of octenyl succinate acetylated starch (OS), gelatin (GE), and tannic acid (TA) were used to fabricate oleogels via an emulsion-template method combined with freeze-drying for algal oil delivery. The results indicated that electrostatic interactions and hydrogen bonding drove the formation of the ternary complex, resulting in a stronger oleogel network and improved viscoelastic properties and storage stability. Under accelerated oxidation, OS-GE-TA oleogels exhibited significantly (p < 0.05) lower peroxide values than OS oleogels, with reductions of 9.55-12.87 meq/kg oil, and effectively inhibited fishy odor-related volatiles. In vitro digestion results showed that the free fatty acid (FFA) release of OS-GE-TA0.4 and OS-GE-TA0.8 oleogels reached 71.17% and 80.42%, respectively, both significantly (p < 0.05) higher than algal oil (64.28%), indicating enhanced lipid digestion and DHA release under simulated gastrointestinal conditions. Overall, the novel oleogels developed in this study have considerable potential for DHA delivery.

PubMedJournal of environmental management2026-07-25

Synergistic microbubble ozonation and CaO2 activation for refinery sludge valorization: Organic carbon liberation and petroleum hydrocarbon degradation.

Shu Di D, Lou Ziyang Z, Yang Kaiyan K, Sun Zhiyi Z et al.

Petroleum-containing waste sludge (PWS) accumulation and internal carbon scarcity are major hurdles in oily wastewater treatment. This study develops a cleaner production strategy using microbubble ozonation (MB-O3) coupled with calcium peroxide (CaO2) activation to simultaneously valorize PWS and remediate hydrocarbons. The MB-O3/CaO2 treatment (initial pH 7.0, CaO2 dosage of 0.25 g/gTS) released 7620 mg/L of soluble chemical oxygen demand (SCOD) and 1163 mg/L of volatile fatty acids (dominated by acetic and propionic acid), improving sludge biodegradability by 6.8-fold. Concurrently, 70.3% total petroleum hydrocarbon (TPH) removal was achieved following pseudo-first-order kinetics, demonstrating broad-spectrum degradation across C10-C40 hydrocarbons. Mechanistic insights reveal that the synergistic effect hinges on the microbubble-enhanced ozone mass transfer and the dual-functional role of CaO2 in sustained H2O2 liberation and self-driven pH modulation. The transition from •O2- dominated alkaline micro-zones to a persistent •OH oxidation regime ensures the synchronous mobilization of entrapped organic carbon and the oxidative cracking of hydrocarbons. Correlation analysis confirms that carbon liberation and TPH removal are mutually reinforcing processes. This strategy facilitates simultaneous waste remediation and carbon liberation, aligning with circular economy principles.

PubMedJournal of hazardous materials2026-07-25

Reactive nitrogen species-driven oxidative and nitrative damage underlying cadmium/mercury co-exposure: A multimodal fluorescent visualization study.

Tan Jiangkun J, Zhang Liangwei L, Wang Yunqing Y, Wang Xiaoyan X et al.

Heavy metal contamination severely threatens ecological environment and human health. Few studies have established an integrated mechanistic framework linking heavy metal co-exposure, reactive nitrogen species (RNS) burst, and progressive cellular injury, especially for multi-metal combined pollution that better reflects real environmental scenarios. As downstream substance of nitric oxide (NO) and representative RNS, peroxynitrite (ONOO-) possess stronger oxidation/nitration capacity compared with hydrogen peroxide (H2O2). The oxidative/nitrifying stress ignited by RNS outburst under cadmium ion (Cd2+) and mercury ion (Hg2+) has not been seemingly clarified yet. Here, we rationally designed a ONOO- fluorescent probe and establish single/co-exposure models to visualize ONOO- dynamics, with the goal of defining a novel RNS-mediated mechanism for Cd2+/Hg2+ synergistic toxicity. Upon addition of ONOO-, the quenching of fluorophore by the recognition group was disrupted, and intramolecular charge transfer (ICT) mechanism restored, showing enhanced fluorescence. The probe has high recognition sensitivity for ONOO- (LOD = 36.4 nM) and good selectivity. Bcy-dpp was employed to observe exogenous and endogenous ONOO- level fluctuation in RAW264.7, HepG2 cells, zebrafish and cockscomb flowers. Our imaging results directly establish a causal link between heavy metal co-exposure, RNS burst, and cellular damage, providing a unified conceptual basis for understanding heavy metal combined toxicology. It is adequately envisioned that our proposed strategy could offer new insights for exploring the toxicology of heavy metal stress.

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