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collagen (Gelfix / Biopad / Condress)

✓ Approved

Merck & Co. · therapeutic agent

What is collagen?

collagen is a therapeutic agent developed by Merck & Co.. It is approved for therapeutic indications via topical.

Drug Profile

Brand NamesGelfix, Biopad, Condress
CompanyMerck & Co.
RouteTopical
StatusApproved

Therapeutic Indications

collagen is developed for 4 unique indications across 3 therapeutic areas.

Therapeutic AreaConditionPhase
General disorders and administration site conditionsImpaired healing✓ Approved
Skin and subcutaneous tissue disordersDiabetic foot✓ Approved
Skin and subcutaneous tissue disordersDecubitus ulcer✓ Approved
Vascular disordersPeripheral venous disease✓ Approved

Related Research Articles

PubMedAdvanced healthcare materials2026-08-25

Engineering Tissue-Relevant Collagen Bundle Architectures via Macromolecular Crowding.

Gong Xiangyu X, Liang Zixie Z, Huang Yaqing Y, Liu Raymond R et al.

Collagen type I forms thick, cell-scaled bundles in native tissues, but standard in vitro collagen gels are composed of disordered thin nanofibrillar networks lacking this architecture. We introduce a simple macromolecular crowding (MMC) strategy, distinct from thermo- or pH-driven gelation, that rapidly assembles collagen into continuous, thick, microscale bundles with tunable dimensions matching healthy and diseased tissue states. These bundles recreate fibrotic and cancer-associated matrix features, enabling direct investigation of how the geometry and topography of collagen fiber networks regulate cell-state transitions and tumor invasion. The method is compatible with collagen from multiple species and collagen-rich decellularized ECM. These single-cell-sized bundles also support robust endothelial sprouting and the formation of aligned microvascular networks in matrices that normally restrict angiogenesis. Bundle suspensions are readily extrudable for bioprinting and injectable delivery applications. Compared to conventional collagen gels, MMC-synthesized bundles are highly tunable to trigger diverse cell behaviors and more accurately replicate native tissue microenvironments, providing a broadly applicable platform for disease modeling and regenerative engineering.

PubMedClinical, cosmetic and investigational dermatology2026-08-25

Multi-Omics Reveals That Hydroxypinacolone Retinoate Alleviates UV-Induced Skin Aging via the p38/MMP9/Collagen Axis.

Wu Haotian H, Zheng Shuiying S, Wen Huihong H, Zou Xiaofang X et al.

UV radiation induces skin photoaging via the p38/MMP9/collagen axis. Hydroxypinacolone Retinoate (HPR) is a retinoic acid precursor with anti‑aging potential, but its concentration-dependent mechanisms remain unclear. This study investigates how HPR protects against UV-induced photoaging through this axis using in vivo experiments combined with transcriptomics and metabolomics. A chronic photoaging model was established in KM mice by UVB irradiation (100 mJ/cm2, every other day for 4 weeks) on the dorsal skin. Topical HPR at concentrations of 0.025%, 0.5%, or 1% was applied immediately after each UV exposure. Histological evaluations included H&E staining for epidermal thickness, Toluidine blue for mast cell infiltration, and Masson's trichrome for collagen deposition. Immunohistochemistry was used to detect collagen I, MMP9, and phosphorylated p38 (p-p38). ELISA measured PIP, TGF-β1, MMP9, TIMP1, elastin, GPX4, ACSL4, and IL-10. Multi-omics analyses (RNA-seq transcriptomics and LC-MS metabolomics) were performed with GO/KEGG enrichment. HPR dose‑dependently improved macroscopic and histological photoaging signs, reducing epidermal hyperplasia and mast cell infiltration while restoring collagen deposition. It upregulated collagen synthesis markers (PIP, TGF‑β1, collagen I) and IL‑10, downregulated MMP9 and p‑p38, corrected the MMP9/TIMP1 balance, preserved elastin, restored GPX4 activity, and suppressed ACSL4. Multi‑omics revealed concentration‑dependent effects: low‑dose HPR affected metabolic pathways, medium‑dose enriched autophagy, and high‑dose triggered immune‑inflammatory rebalancing. HPR alleviates photoaging through coordinated mechanisms: promoting collagen synthesis, inhibiting MMP9‑mediated degradation, preserving elastin, reducing oxidative stress and ferroptosis, and shifting inflammation toward repair via IL‑10, while suppressing the p38/MMP9 axis. The dose‑dependent hormetic response-metabolic adaptation at low dose, autophagy at medium dose, immune modulation at high dose-supports HPR as a multi‑target agent for skin rejuvenation.

PubMedSmall (Weinheim an der Bergstrasse, Germany)2026-08-25

Tungsten Oxide Sub-Nanowires as Inorganic Dental Collagen Crosslinker With Antibacterial Capacity.

Liu Zhiyi Z, Shen Xu X, Li Pingping P, Shi Wenxiong W et al.

The structural stability of dental collagen plays a critical role in preserving the integrity and function of tooth. However, conventional organic cross-linking agents are often compromised in moist environments and vulnerable to bacterial degradation. Here, we propose an inorganic synergistic "cross-linking-antibacterial" strategy utilizing tungsten oxide sub-nanowires (TO-SNWs). This strategy effectively overcomes the limitations of organic cross-linking imposed by humid environments and significantly enhance the thermostability, mechanical strength, and structural stability of dental collagen. Furthermore, TO-SNWs exert targeted antibacterial activity against the Firmicutes phylum, which harbors a majority of oral pathogens, while maintaining the overall diversity of the oral microbiota. Mechanism analysis reveals that antimicrobial effects stem from multilevel TO-SNWs-bacteria interactions, which compromise the permeability and metabolism. In conclusion, this inorganic "cross-linking-antibacterial" approach not only reinforces dental collagen stability, but also suppresses pathogenic microorganisms' activity, representing a promising and innovative strategy for dentin biomodification.

PubMedERJ open research2026-08-25

Lung fibroblast activation protein expression is associated with extracellular matrix remodelling in post-acute sequelae of SARS-CoV-2.

Postma Niels J NJ, van Leer Bram B, Simões Filipa B FB, van der Velde Petra P et al.

Pulmonary fibroblast activation protein expression in post-acute sequelae of SARS-CoV-2 is associated with circulating extracellular matrix fragments reflecting collagen type III degradation (C3M) and collagen type VI formation (PRO-C6) https://bit.ly/47rLr0G.

PubMedInternational journal of implant dentistry2026-08-25

Prefabricated CAD/CAM zirconia membrane versus contour augmentation with early implant placement in the anterior maxilla: a randomized controlled clinical trial.

Anas Mohammed Mashhout MM, Gabr Amr Mostafa AM, El-Makaky Yasser Y, Helal Mohamed H MH

Horizontal ridge deficiencies in the anterior maxilla frequently compromise ideal implant positioning and esthetic outcomes. This randomized controlled clinical trial evaluated the clinical performance of customized CAD/CAM zirconia membranes compared with conventional collagen membranes for guided bone regeneration (GBR) performed simultaneously with early implant placement. Twenty systemically healthy patients requiring single-tooth replacement in the anterior maxilla underwent early implant placement (4-8 weeks after tooth extraction) combined with simultaneous contour augmentation using a composite graft of autogenous bone and deproteinized bovine bone mineral (1:1). Participants were randomly assigned to receive either a customized CAD/CAM zirconia membrane (test group, n = 10) or a double-layer collagen membrane (control group, n = 10). The primary outcome was horizontal bone gain (HBG) at 6 months, assessed by standardized CBCT superimposition. Secondary outcomes included soft tissue healing evaluated using the Modified Wound Healing Index (MWHI), membrane exposure (wound dehiscence), and patient-reported postoperative pain assessed using a visual analogue scale (VAS). All implants survived during the 6-month follow-up period (100% survival rate). Mean HBG was 6.18 ± 0.60 mm in the test group and 5.85 ± 0.37 mm in the control group, with no statistically significant difference between groups (p = 0.158). Membrane exposure occurred in all patients receiving customized zirconia membranes (10/10, 100%), whereas no membrane exposure was observed in the collagen membrane group (0/10, 0%). Despite the higher exposure rate, graft stability and horizontal bone regeneration were not adversely affected. Soft tissue healing was significantly more favorable in the collagen membrane group. Postoperative pain decreased significantly over time in both groups, with no significant intergroup differences. Customized CAD/CAM zirconia membranes achieved horizontal bone augmentation comparable to conventional collagen membranes when used for simultaneous GBR during early implant placement. Although customized zirconia membranes were associated with a substantially higher membrane exposure rate, regenerative outcomes and implant survival remained unaffected, indicating that customized rigid barriers may represent a predictable alternative for horizontal ridge augmentation while offering potential advantages in surgical precision and procedural efficiency. ClinicalTrials.gov NCT06987149.

PubMedRSC advances2026-08-25

Crosslinking strategies govern the morphology and biological performance of decellularized extracellular matrix particle-tyramine-modified hyaluronic acid hydrogels.

Xu Jiangyao J, Wychowaniec Jacek K JK, D'Este Matteo M, Jiang Nan N et al.

Decellularized extracellular matrix (dECM) particle-based hydrogels hold promise for cartilage repair. Due to its chondropermissive properties, tuneable viscoelastic profile, and the potential to form covalent bonds with ECM proteins, tyramine-modified hyaluronic acid (THA) is an ideal candidate as a binder for dECM particles. However, the incorporation of the dECM particles into a viscoelastic hydrogel may interfere with its crosslinking. The goal of this study was to compare two crosslinking methods: (1) horseradish peroxidase/Eosin (HRP/Eo) with H2O2 and (2) sodium persulfate/ruthenium (SPS/Ru) for fabricating THA hydrogels containing dECM particles, and to investigate their biological and biomechanical effect for cartilage tissue engineering. Incorporation of dECM markedly enhanced the mechanical properties of the composites in both crosslinking methods, increasing the storage modulus by 10-fold, while leaving the linear viscoelastic region unchanged. Chondrocyte-laden dECM hydrogels demonstrated sustained cytocompatibility over 14 days, evidenced by stable DNA content throughout the culture period. Compared with SPS/Ru, hydrogels showed more effective incorporation of dECM particles into the HRP/Eo-crosslinked network, as indicated by a lower I ∼1630/I ∼1030 ratio from FTIR analysis, clear morphological changes in confocal microscopy, higher glycosaminoglycan (GAG) and collagen retention, as well as slower GAG and collagen release. HRP/Eo-dECM hydrogels promoted stronger gene expression of type II collagen (COL2) and higher compressive modulus compared with SPS/Ru-dECM hydrogels. In summary, HRP/Eo crosslinked dECM hydrogels demonstrated improved mechanical performance and chondrocyte redifferentiation effect compared to SPS/Ru, attributed to the difference in hydrogel morphology. These findings identify the HRP/Eo crosslinking method as a favorable strategy for engineering dECM particle-THA hydrogels for cartilage regeneration.

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